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A generous $25 million commitment from the Once Upon a Time Foundation will create the Raynor Cerebellum Project at UT Southwestern Medical Center (RCP-UTSW) to investigate diseases associated with cerebellum dysfunction, with the goal of discovering how to preserve and restore lost brain function.

Summertime trips to lakes or pools to escape the heat can sometimes lead to ear infections caused by excess moisture in the ear canal.

UT Southwestern sleep researcher Masashi Yanagisawa, M.D., Ph.D., has been awarded the Breakthrough Prize in Life Sciences for his discovery of the cause of narcolepsy.

Fifteen scientists in the Harold C. Simmons Comprehensive Cancer Center at UT Southwestern received more than $17 million in research funds in the latest round of grants awarded by the Cancer Prevention and Research Institute of Texas (CPRIT).

Oxygen-deprived newborns who undergo cooling therapy to protect their brains are at an elevated risk of seizures and brain damage during the rewarming period, which could be a precursor of disability or death

If you have resolved to eat healthier to manage your diabetes, blood pressure, or cholesterol, parties can present a challenge, so it’s important to have a game plan before tackling the Super Bowl spread.

Patients with both a high lipoprotein(a) and high coronary artery calcium score (CAC) face a more than 20% higher risk of heart attack or stroke over the following 10 years, according to findings from a multicenter study led by preventive cardiologists at UT Southwestern Medical Center.

Mechanical hearts spur some regeneration in dormant parts of failing hearts, according to a UT Southwestern pilot study that shows promise for developing regenerative heart therapies.

A new approach that targets the cellular machinery that viruses need to reproduce – rather than the virus itself – appears to stem replication of a common childhood pathogen known as respiratory syncytial virus (RSV), UT Southwestern researchers report in a new study.

UT Southwestern researchers report the first structural confirmation that endogenous – or self-made – molecules can set off innate immunity in mammals via a pair of immune cell proteins called the TLR4-MD-2 receptor complex.