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A singer’s primary genre can impact the likelihood of developing vocal fold injury and may even influence the specific type of injury that occurs, a recent study by UT Southwestern researchers suggests.

A mutation in the gene that causes cystic fibrosis may accelerate heart function decline in those with Duchenne muscular dystrophy (DMD), a new study by UT Southwestern researchers suggests.

Complications after an amputation left Scott Bryson, a 50-year-old father of three, unable to walk. But in June 2023, he took his first steps in nearly a decade after receiving a revolutionary surgical procedure at UT Southwestern Medical Center.

Grouped by specialty, here are the matches achieved by members of the UT Southwestern Medical School Class of 2019.

Revised DFW model shows a dramatic spike in DFW hospitalizations related to COVID, predicting a potential 20% surge over the next two weeks.

In Memoriam: David J. Mangelsdorf, Ph.D., pioneer in orphan nuclear receptor research and Chair of Pharmacology Published on: August 09, 2025 • By: Newsroom Share Share on Facebook Share on Twitter Share on LinkedIn Email this page Print this page DALLAS – Aug. 9, 2025 – David J. Mangelsdorf, Ph.D., Chair and Professor of Pharmacology at UT Southwestern Medical Center, a visionary scientist who pioneered the once-obscure realm of orphan nuclear receptors (a class of proteins involved in gene

Anesthesiologists are exploring the use of hypnosis through virtual reality to lessen postoperative pain and anxiety in children.

Faculty, family, and friends gathered on the basketball court and watched as more than 200 graduating UT Southwestern medical students joined thousands more at medical schools across the country to reveal their futures.

Millions of Americans have chronic shoulder pain, which can have a significant impact on quality of life. Although some get relief from medications, physical therapy, or joint injections, those with persistent pain often pursue more aggressive treatments such as surgeries and permanent

Genetic mutations that accumulate in the adult liver that can promote regeneration in the context of chronic liver damage.