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Researchers at UT Southwestern have uncovered evidence that the higher prevalence of “malignant” enlargement of the heart among blacks contributes to the higher incidence of heart failure in this population.

Three decades-old antibiotics administered together can block a type of pain triggered by nerve damage in an animal model, UT Southwestern researchers report.

More teenage athletes experienced depression and anxiety during the early weeks of the pandemic, when COVID-19 restrictions curtailed sports activities, according to a survey of 600 child and adolescent athletes led by researchers at UT Southwestern and Scottish Rite for Children.

The Centers for Medicare & Medicaid Services (CMS) announced that the Southwestern Health Resources Accountable Care Network (SWHR) saved more than $37 million in 2018.

A UT Southwestern research team has catalogued gene activity in the skeletal muscle of mice, comparing healthy animals to those carrying a genetic mutation that causes Duchene muscular dystrophy (DMD) in humans.

James J. Collins III, Ph.D., Associate Professor of Pharmacology at UT Southwestern Medical Center who leads groundbreaking research into the parasitic disease schistosomiasis, has been named a Howard Hughes Medical Institute (HHMI) Investigator.

A study of gene activity in the brain’s hippocampus, led by UT Southwestern researchers, has identified marked differences between the region’s anterior and posterior portions.

A UT Southwestern research team has generated biological structures that resemble blastocysts, the structures that form from the early development of fertilized eggs in mammals, using previously established human embryonic stem cells derived from embryos donated for research and human-induced

Expanded telehealth services at UT Southwestern have proved effective at safely delivering patient care during the pandemic, leading to an increase in patients even in specialties such as plastic surgery, according to a new study.

Loss of an enzyme necessary for a process called lipoylation disrupts the way cancer cells copy their DNA, increasing their vulnerability to a class of anticancer drugs known as PARP inhibitors, a study led by UT Southwestern Medical Center researchers shows.