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UT Southwestern researchers find that humans and mice with pancreatitis are deficient in a stress hormone called FGF21.

Dr. Stephanie Tow, M.D., completed her first week of providing volunteer care at the Tokyo 2020 Olympics.

Researchers with the Peter O’Donnell Jr. Brain Institute at UT Southwestern have identified a new protein implicated in cell death that provides a potential therapeutic target that could prevent or delay the progress of neurodegenerative diseases following a stroke.

A study of gene activity in the brain’s hippocampus, led by UT Southwestern researchers, has identified marked differences between the region’s anterior and posterior portions.

Restricting calories has long been recognized as a powerful way to live longer, with periods of intermittent fasting proving more effective than a steady diet. However, the mechanism behind this phenomenon has been unclear.

New national guidance for managing cholesterol and other blood lipids calls for earlier risk assessment, more personalized treatment, and renewed emphasis on long-term cardiovascular prevention – changes experts say could significantly reduce heart attacks and strokes over a lifetime.

Neurons in a part of the brain known as the ventromedial hypothalamus (VMH) appear to direct the body to boost endurance in response to exercise, a study co-led by researchers at UT Southwestern Medical Center shows.

The U.S. Food & Drug Administration has expanded the approved use of belzutifan for treatment of metastatic kidney cancer, another milestone for the novel, first-in-class kidney cancer drug arising from scientific discoveries at UT Southwestern Medical Center.

Funded by a Specialized Program of Research Excellence (SPORE) award from the National Cancer Institute (NCI), the Kidney Cancer Program (KCP) at UT Southwestern’s Harold C. Simmons Comprehensive Cancer Center reports the largest and most diverse catalog of kidney cancer tumor models to date.

Activating specific neurons in a part of the brain that serves as the body’s master circadian pacemaker caused mice to eat significantly more during a time of day when they would normally be at rest, a UT Southwestern Medical Center study shows.